The bands on this page
| Result | What it is called here |
|---|---|
| under 0.5 | Excellent |
| 0.5 to 0.6 | Good |
| 0.6 to 0.8 | Elevated risk |
| over 0.8 | High risk |
What goes in, and where each number comes from
Every input is a real test. Follow one to see its normal range, its optimal range and what moves it — the calculation is only as good as the numbers you feed it.
What it costs to actually get these numbers
| The 2 tests on their own | $44.10 |
| The cheapest panel that covers them — Real Cardiovascular Risk (9 markers) | $187.60 |
Prices are Marek Diagnostics with the 10% code already applied, and they are what was filed when this page was built — check the cart. Ordering through these links pays a commission at no cost to you.
What could have moved the measurement, not the marker
A wrong number and a real change look identical on a report. These are the reasons a result here can be an artifact of how the blood was drawn or run, and they are worth ruling out before anything else.
Less affected by eating than a calculated LDL, which is one of its advantages — but it still shifts modestly.
What to do: Fasted is preferable; consistency matters more.
All lower ApoB, which is the point. The interference is in interpretation — a good ApoB on treatment is not the same as a good untreated one.
What to do: Record what you were taking at each draw.
Testosterone lowers ApoA1; estrogen raises it. Both are real drug effects rather than changes in cardiovascular risk of the same size.
What to do: Record your hormone status on the result — otherwise a low ApoA1 on TRT reads as a risk finding rather than a drug effect.
ApoB falls during acute illness along with the rest of the lipid panel, so a post-admission draw understates your true particle count.
What to do: Wait six weeks after anything significant.
1 more interferences are listed on the marker pages themselves: ApoB (Apolipoprotein B) · Apolipoprotein A-1.